Normal CXR
- measured inside ribs- PA, inspiratory, erect film
~ 1.5 cm change in diameter between inspiration / expiration
"Apparantly" Normal CXR
- clavicles & hyperparathyroidism- spine
- small pneumothorax- apical lung disease
Respiratory Distress & "Normal" CXR
- FB, secretions, ETT cuff, epiglottis, croup, etc. Single - Common Multiple - Common Single - Uncommon Multiple - Uncommon
Features suggestive of malignancy, CXR appearances Lobar pneumonia
2. no "air" bronchogram Common causes Bronchopneumoinia Butterfly Appearance Reversed Butterfly Appearance Bilateral Hilar Enlargement lymphadenopathy
- renal cell- melanoma- nead/neck carcinoma
vascular
pulmonary hypertension- chronic lung disease
- multiple pulmonary emboli- primary pulmonary hypertension
post-pulmonary stenosiscongenital large pulmonary arteryaneurysm
Cavitating Lesion on CXR Lung Infections
- Staph., gram negatives, Klebsiella, anaerobes
- Staph., gram negatives, Klebsiella, anaerobes, fungi
- viral, TB, atypical, fungal, pneumocystis
Apical Disease on CXR Peribronchial Thickening Diffuse Pulmonary Infiltrates NB: divide into acute or chronic, chronic upper or lower distribution acute
- sepsis, trauma, transfusion reaction, fat emboli, etc.
- contusions, trauma- infarction, Goodpasture's, coagulopathy- idiopathic haemosiderosis
in severe COAD the distribution of oedema is patchy subacute chronic: upperzones
ankylosing spondylitis, ulcerative colitis
chronic: lowerzones
amiodarone, bleomycin, busulphan, methysergide, hydrallazine, procainamide,sulphonamides
Upper Lobe → SCHART Lower Lobe → RASIO
- other- busulphan, bleomycin, amiodarone, methotrexate
Diffuse Interstitial Disease + Mediastinal Lymphadenopathy Diffuse Interstitial Disease + Skeletal Abnormality
- RA, scleroderma, sarcoidosis- hypertrophic pulmonary osteoarthropathy
Miliary Opacities Cardiophrenic Angle Mass Hyperinflated Lungs bilateral hyperinflation unilateral hyperinflation apparent unilateral hyperinflation Pulmonary Oligaemia Pulmonary Plethora
- polycythaemia- thyrotoxicosis- fluid overload
- ASD, VSD, PDA- partial anomalous pulmonary venous drainage
- transposition, truncus arteriosus- partial anomalous pulmonary venous drainage
Massive Lesion On CXR > 6 cm Mediastinal Masses commonest anterior middle posterior Calcification on CXR NB: not carcinoma, not hydatid localised calcification diffuse calcification hilar calcification
tuberculosissilicosissarcoidosishistoplasmosis
"eggshell" calcification pleural calcification Extrapleural Mass on CXR Pleural Effusion NB: > 300 ml
homogenous opacity obscures heart border and diaphragm
increase distance between lung and stomach gas on left. lateral decubitus reveals change in meniscus and useful for small or unusual effusion Massive Pleural Effusion Rib Notching Aspiration Pneumonitis COAD / CAL Sarcoidosis
opacities spreading from hilum into parenchyma
Tuberculosis
primary lesion in middle lobe or apex of lower lobe
Carcinoma lung metastases central mass peripheral mass → i. Pulmonary Embolus
transient unilateral increase in lung lucency
pleurally-based, wedge-shaped or 'D'-shaped opacity on lateral CXR
NB: lower lobe more common than upper lobe; right lung more common than left lung Pulmonary Arterial Hypertension
enlarged, well-defined hilar vessels - arise from hilum
CXR changes are late Pulmonary Venous Hypertension NB: ↑ magnification on portable AP films, ∴ may exceed these limits
Fat Embolus Lung Infiltrates In Renal Failure Mesothelioma / Asbestosis Def'n: asbestosis Silicosis Heart Size small heart massive enlargement moderate enlargement
- supine or AP film- raised hemidiaphrams
↑ pulmonary blood flow & LA volume overload
Cardiac Failure CCF
"cotton-wool" opacities around bronchi
pleural effusion, fluid in lobar fissures
NB: severe CAL
interstitial & vascular changes may not occur
lung fields reflect cardiac function better than heart size does
Pulmonary Oedema chronic / cardiogenic acute and/or non cardiogenic
no lymphatics nor venous congestion visible
NB: Kerley's lines: A & B →
large, 4-6 cm, irregular, radiate from hilum to upper lobes
short 1-2 cm, horizontal, basal, touch pleural margin
permanent - MS, tumour, lymphangitis, pneumoconioses
fine curvillinear, often generalised, giving reticular pattern
Pulmonary Oedema: Acute NB: signs usually present in acute pulmonary oedema, NB: all but #3 may be present in acute cardiogenic pulmonary oedema Pulmonary Oedema: Unilateral
re-expansion of collapsed lung or pneumothorax
Mitral Stenosis
straight left heart border & ↑ LA appendage
normal heart size in uncomplicated cases Left Atrial Dilatation Fallot's Tetralogy
apex of the heart raised above level of hemidiaphragm
Atrial Septal Defect ASD
septal defect with mongolism can → isolated RUL congestion (mechanism unknown)
NB: ↑↑ pulmonary blood flow & RV output with volume overload Eisenmenger's Syndrome Def'n: reversal of right → left shunt as a result of pulmonary hypertension Coarctation of the Aorta
"wasting" or "3-sign" on descending aorta
Pericardial Effusion NB: XRay changes late
"water-bag" cardiomegaly, large globular cardiac shadow
acute angle between cardiac shadow and hemidiaphragms.
- no movement of heart seen to blurr film
Constrictive Pericarditis
chronic idiopathicchronic renal failurerheumatoid arthritisneoplastictuberculosisirradiation
Patent Ductus Arteriosus Right Heart Failure no alveolar oedema SXR
focal loss of bone density lining pituitary fossa
> 2 mm shift of calcified pineal on lateral film
CT Scan
loss of grey-white matter differentiation
Hydrocephalus CT Scan Criteria
± enlargement of basal cisterns and 4th ventricle
± periventricular deceased density → communicating hydrocephalus
may have associated intracerebral blood or oedema
convex (biconvex) bulging opaque swelling
classical shape does not occur post-surgically
associated oedema and midline shift common
often associated intracerebral haemorrhages
area of decreased density within brain substanceusually within the territory of a major vesselreduced density & mild mass effect may be seen as early as 6 hrs, usually > 24 hrs
may be focal or generalized, with loss of grey/white matter differentiation may be normal in the presence of marked oedema & raised ICP
low density lesion which has peripheral enhancement on contrast
the majority have no CT abnormalitiesHSV characteristically results in bilateral (initially unilateral) reduction in densityand surrounding compression of the temporal poles
basal ganglia may appear more distinct due to ↑ contrast
the majority have no CT abnormalitiesmeningeal enhancement may be seen with contrast
AXR Review
liver edge, spleen edge, psoas margins, renal outlines
- peritoneal cavity- biliary tree- renal system- uterus- subcutaneous
- bodies, disc spaces, transverse processes
Paralytic Ileus
increased air fluid levels predonimantly in small bowel
no signs of mechanical obstruction (hernia, volvulus)
Small Bowel Obstruction
many plica semilunares visible
gas below inguinal ligament NB: gas in biliary tree + SBO at Mekel's Large Bowel Obstruction Plain AXR
- peripheral with haustra > 5 cm
SBO usually absent if ileocaecal valve competent
NB: if in doubt, barium enema will exclude pseudo-obstruction Ischaemic Bowel Plain AXR
small bowel, ascending and transverse colon
Portal Venous Gas NB: distribution
- peripheral to within 2 cm of liver edge
Pneumothorax CXR
translucent area superiorly without lung markings
Pneumothorax Aetiology Pulmonary Barotrauma
linear air streaks radiating towards hilum
Pulmonary Contusion
immediate (< 6 hrs) ill-defined density or consolidation
Lung Infiltrates Post-Traumatic Pneumomediastinum
does not extend beyond pericardial reflectionsgas beneath heart
Thoracic Aortic Rupture highly suggestive
left bronchus displaced inferiorly low association Diaphragmatic Rupture
CXR often normal, ∴ with signs of major chest injury → high suspicion
gastric dilatationhigh hemidiaphragmloculated pbeumothoraxsubpulmonic haematoma
Oesophageal Rupture Raised Hemidiaphragm
- abscess, ascites, pancreatitis, hepatomegaly, tumour- obesity, pregnancy
Rheumatoid Arthritis
- but not DIP
Lijst van verboden farmacologische groepen van stoffen en verboden methoden (“de dopinglijst”) I. Verboden groepen van stoffen A. Stimulantia B. 1. androgene anabole steroïden 2. ß2-agonisten D. Diuretica E. II. Verboden methoden A. Bloeddoping B. Toediening van kunstmatige zuurstofdragers of middelen die het plasmavolume vergroten Farmacologische, chemische en f
Robert V. Kolbusz, M.D. Why are you here today? (If you are not here for Acne or a Rash, fill out as many questions as you can from this Questionnaire.) Please DO NOT MARK ON ANY UNUSED QUESTIONNAIRES 3825 Highland • Suite 5C • Downers Grove, IL 60515 • 630-964-2000 Fax 630-964-2033 Robert V. Kolbusz, M.D. ACNE QUESTIONNAIRE PATIENT :________________________